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Table 1.

CHI3L1/YKL-40-associated genes

GeneFold change
CellsTumors
Ovalbumin 72.8 1.5 
Nicotinamide N-methyltransferase 11.3 4.1 
Collagen, type VI, α1 4.7 1.7 
Collagen, type VI, α2 4.7 2.8 
Tenascin C (hexabrachion) 3.9 2.4 
Basic helix-loop-helix domain containing, class B, 2 2.4 1.7 
Epithelial membrane protein 1 2.4 1.6 
Insulin-like growth factor binding protein 5 2.3 2.2 
Collagen, type XVI, α1 2.1 2.2 
Likely orthologue of rat brain specific binding protein/Ras-induced senescence 1 1.9 2.1 
Transforming growth factor-β receptor II 1.8 1.6 
Vascular endothelial growth factor 1.9 2.6 
Transforming growth factor β-induced 1.8 2.5 
A kinase (PRKA) anchor protein 11 1.7 1.9 
Cadherin 11, type 2, OB-cadherin (osteoblast) 1.7 1.8 
Pre-B-cell colony-enhancing factor 1.7 4.5 
ADP-ribosylation factor-like 7 1.6 1.7 
KIAA0062 protein 1.6 1.7 
Caldesmon 1 1.5 1.8 
Plasminogen activator inhibitor type 1 1.5 2.6 
Paternally expressed 10 0.5 0.4 
GeneFold change
CellsTumors
Ovalbumin 72.8 1.5 
Nicotinamide N-methyltransferase 11.3 4.1 
Collagen, type VI, α1 4.7 1.7 
Collagen, type VI, α2 4.7 2.8 
Tenascin C (hexabrachion) 3.9 2.4 
Basic helix-loop-helix domain containing, class B, 2 2.4 1.7 
Epithelial membrane protein 1 2.4 1.6 
Insulin-like growth factor binding protein 5 2.3 2.2 
Collagen, type XVI, α1 2.1 2.2 
Likely orthologue of rat brain specific binding protein/Ras-induced senescence 1 1.9 2.1 
Transforming growth factor-β receptor II 1.8 1.6 
Vascular endothelial growth factor 1.9 2.6 
Transforming growth factor β-induced 1.8 2.5 
A kinase (PRKA) anchor protein 11 1.7 1.9 
Cadherin 11, type 2, OB-cadherin (osteoblast) 1.7 1.8 
Pre-B-cell colony-enhancing factor 1.7 4.5 
ADP-ribosylation factor-like 7 1.6 1.7 
KIAA0062 protein 1.6 1.7 
Caldesmon 1 1.5 1.8 
Plasminogen activator inhibitor type 1 1.5 2.6 
Paternally expressed 10 0.5 0.4 

NOTE: In vivo and in vitro expression patterns for cells overexpressing CHI3L1/YKL-40 were compared. In vivo expression patterns were derived from glioblastoma tumor samples that were divided into two groups based on CHI3L1/YKL-40 expression levels, whereas in vitro profiles were obtained with CHI3L1/YKL-40 and mock-transfected cells. Genes that differed by 1.5-fold in these comparisons and were common to both in vitro and in vivo profiles are shown.

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