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Targeting VDAC2 Induces Inflammation and Immune-Mediated Tumor Cell Killing
April 3, 2025
Major Finding: VDAC2 blocks IFNγ-mediated tumor cell killing and anticancer inflammatory immune reprogramming.
Concept: Mitochondrial DNA release and subsequent STING activation mediate VDAC2 loss–induced antitumor inflammatory phenotypes.
Impact: VDAC2 is a promising therapeutic target that may enhance the anticancer efficacy of immunotherapies.
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