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Su Sien Ong
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Journal Articles

Targeting Histone Demethylases in MYC-Driven Neuroblastomas with Ciclopirox

Jun Yang, Sandra Milasta, Dongli Hu, Alaa M. AlTahan, Rodrigo B. Interiano, Junfang Zhou, Jesse Davidson, Jonathan Low, Wenwei Lin, Ju Bao, Pollyanna Goh, Amit C. Nathwani, Ruoning Wang, Yingdi Wang, Su Sien Ong, Vincent A. Boyd, Brandon Young, Sourav Das, Anang Shelat, Yinan Wu, Zhenmei Li, Jie J. Zheng, Ashutosh Mishra, Yong Cheng, Chunxu Qu, Junmin Peng, Douglas R. Green, Stephen White, R. Kiplin Guy, Taosheng Chen, Andrew M. Davidoff
Journal: Cancer Research
Cancer Res (2017) 77 (17): 4626–4638.
https://doi.org/10.1158/0008-5472.CAN-16-0826
Published: 31 August 2017
View articletitled, Targeting Histone Demethylases in MYC-Driven Neuroblastomas with Ciclopirox
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Includes: Supplementary data
Journal Articles

Multikinase Inhibitors Induce Cutaneous Toxicity through OAT6-Mediated Uptake and MAP3K7-Driven Cell Death

Eric I. Zimmerman, Alice A. Gibson, Shuiying Hu, Aksana Vasilyeva, Shelley J. Orwick, Guoqing Du, Gerard P. Mascara, Su Sien Ong, Taosheng Chen, Peter Vogel, Hiroto Inaba, Michael L. Maitland, Alex Sparreboom, Sharyn D. Baker
Journal: Cancer Research
Cancer Res (2016) 76 (1): 117–126.
https://doi.org/10.1158/0008-5472.CAN-15-0694
Published: 03 January 2016
View articletitled, Multikinase Inhibitors Induce Cutaneous Toxicity through OAT6-Mediated Uptake and MAP3K7-Driven Cell Death
Open the PDF for Multikinase Inhibitors Induce Cutaneous Toxicity through OAT6-Mediated Uptake and MAP3K7-Driven Cell Death in another window
Includes: Supplementary data
Journal Articles

Abstract 4415: Tyrosine phosphorylation is essential for OCT2 function and a target of inhibition by TKIs

Navjotsingh Pabla, Jason A. Sprowl, Su Sien Ong, Alice A. Gibson, Guoqing Du, Wenwei Lin, Shuiying Hu, Lie Li, Taosheng Chen, Alex Sparreboom
Journal: Cancer Research
Cancer Res (2015) 75 (15_Supplement): 4415.
https://doi.org/10.1158/1538-7445.AM2015-4415
Published: 01 August 2015
View articletitled, Abstract 4415: Tyrosine phosphorylation is essential for OCT2 function and a target of inhibition by TKIs