In the review on RASSF1 tumor suppressor gene in the May 1, 2005 issue of Cancer Research (1), there was an error in Fig. 1. The correct Fig. 1 appears below. Also, in the Addendum, the reference should have read as follows: Tommasi S, Dammann R, Zhang Z, Wang Y, Liu L, Tsark WM, Wilczynski SP, Li J, You M, Pfeifer GP. Cancer Res 2005;65:92–8.

Figure 1.

Transcription map of the RASSF1 gene locus in 3p21.3. RASSF1A to RASSF1G are generated by differential promoter usage (arrows) and alternative splicing. The positions of promoter associated CpG islands (black lines) as predicted by the UCSC Genome Browser May 2004. The domain structure of the protein products (predicted using Prosite): C1, DAG-binding domain; RA, RalGDS/AF6 Ras association domain; and SARAH, Sav/RASSF/Hpo interaction domain. ATM is the ATM-kinase phosphorylation consensus sequence (11). Position of the extra 4aa in RASSF1D and RASSF1E (white).

Figure 1.

Transcription map of the RASSF1 gene locus in 3p21.3. RASSF1A to RASSF1G are generated by differential promoter usage (arrows) and alternative splicing. The positions of promoter associated CpG islands (black lines) as predicted by the UCSC Genome Browser May 2004. The domain structure of the protein products (predicted using Prosite): C1, DAG-binding domain; RA, RalGDS/AF6 Ras association domain; and SARAH, Sav/RASSF/Hpo interaction domain. ATM is the ATM-kinase phosphorylation consensus sequence (11). Position of the extra 4aa in RASSF1D and RASSF1E (white).

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1
Agathanggelou A, Cooper WN, Latif F. Role of the Ras-association domain family 1 tumor suppressor gene in human cancers.
Cancer Res
2005
;
65
:
3497
–508.